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β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2 and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK pathway - ScienceDirect
ATP accelerates LPS-induced NO production and iNOS expression. (A) NO... | Download Scientific Diagram
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Titanium dioxide nanoparticles enhance macrophage activation by LPS through a TLR4-dependent intracellular pathway - Toxicology Research (RSC Publishing)
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Regulation of NO production from macrophages by lipopolysaccharide and... | Download Scientific Diagram
Protective role of endothelial calpain knockout in lipopolysaccharide-induced acute kidney injury via attenuation of the p38-iNOS pathway and NO/ROS production | Experimental & Molecular Medicine
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Lipopolysaccharide (LPS)-induced nitric oxide (NO) production and... | Download Scientific Diagram
Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock | Cell Research
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Bavachin attenuates LPS-induced inflammatory response and inhibits the activation of NLRP3 inflammasome in macrophages - ScienceDirect
LPS induces the expression of iNOS and the production of NO. The... | Download Scientific Diagram
Effect of EMM on LPS-induced NO and PGE2production in RAW 264.7 cells.... | Download Scientific Diagram
Figure 4 from Antagonistic effects of acetylshikonin on LPS-induced NO and PGE2 production in BV2 microglial cells via inhibition of ROS/PI3K/Akt-mediated NF-κB signaling and activation of Nrf2-dependent HO-1 | Semantic Scholar
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Acetate Suppresses Lipopolysaccharide-stimulated Nitric Oxide Production in Primary Rat Microglia but not in BV-2 Microglia Cells | Bentham Science
Modulation of LPS stimulated NF-kappaB mediated Nitric Oxide production by PKCε and JAK2 in RAW macrophages | Journal of Inflammation | Full Text
